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. 2021 May 14;12:648688. doi: 10.3389/fphar.2021.648688

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Copyright © 2021 Mou, Zhou, Feng, Zhang, Lin, Aiyasiding, Li, Ding, Liao, Bian and Tang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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LIQ treatment suppressed LPS-induced oxidative stress and apoptosis in vivo. (A) Mice were treated with LPS (30 mg/kg, intraperitoneal injection) for observing 48 h to draw survival curves (n = 20), (B) TUNEL staining was performed to examine cellular apoptosis in mouse hearts (n = 6), (C) Calculating the TUNEL positive nucleus (n = 6), (D) Representative western blots of BAX, Bcl-2 and GAPDH, (E,F) relative quantitative results of BAX/GAPDH and Bcl-2/GAPDH (n = 6). *p < 0.05 compared with the indicated group, the data were expressed as the Mean ± SEM and were compared by one-way ANOVA with Tukey post hoc analysis, A log-rank test was used to compare survival curves.