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. 2021 Mar 5;320(5):H1822–H1835. doi: 10.1152/ajpheart.00673.2020

Table 2.

Results for maximum developed tension (Tdev) at pCa 4.8

Tdev (kPa) NTGβ NTGβ,PKA t/tβ t/tβ,PKA ANOVA
n 9 8 10 10
Pi (mM) = 0 25.33 ± 1.60 14.98 ± 2.37 17.52 ± 1.76 13.38 ± 1.26 Main effects
1 21.49 ± 1.21 13.90 ± 2.41 16.34 ± 1.64 12.67 ± 1.11 *MYBPC3, †Pi, †PKA
2 20.34 ± 1.01 13.14 ± 2.23 15.10 ± 1.45 11.68 ± 1.03
3 19.20 ± 1.03 11.98 ± 2.07 13.87 ± 1.27 10.66 ± 0.92 Interactions
4 18.38 ± 0.80 11.23 ± 1.89 13.05 ± 1.19 9.90 ± 0.91 *Pi×PKA
5 17.74 ± 0.72 10.53 ± 1.71 12.36 ± 1.20 9.09 ± 0.79
8 16.37 ± 0.43 9.22 ± 1.46 10.72 ± 1.11 7.63 ± 0.72
12 14.18 ± 0.73 7.88 ± 1.22 9.05 ± 1.01 6.02 ± 0.68
ΔTdev/ΔPi −1.38 ± 0.17 −0.90 ± 0.18 −1.05 ± 0.16 −0.88 ± 0.14 *PKA

Values are means ± SE; n, number of strips. Statistical results reflect repeated-measures ANOVA and demonstrate statistical significance for the main effects of MYBPC3, Pi, PKA, and for interactions Pi × MYBPC3, PKA × MYBPC3, Pi × PKA, Pi × PKA × MYBPC3. The sensitivity of Tdev to Pi was calculated by linear regression using values measured at 0–5 mM Pi and denoted here as ΔTdev/ΔPi. Two-way ANOVA was then used to determine whether ΔTdev/ΔPi was influenced by the absence of cMyBP-C and/or PKA. *P < 0.05, †P < 0.01. NTGβ, nontransgenic; t/tβ, transgenic lacking cMyBP-C; PKA, protein kinase A.