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. 2021 May 31;11:11372. doi: 10.1038/s41598-021-90779-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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CTRP9 overexpression improved endothelial function in pulmonary arteries from HPH rats. (A) Dose–response curves for ACh-induced relaxation were obtained from pulmonary arteries of different groups (n = 6). Data are expressed as the percentage of the contraction to phenylephrine (PE). (B) Dose–response curves for SNP-induced relaxation were obtained from pulmonary arteries of different groups (n = 6). Data are expressed as the percentage of the contraction to PE. Data are the mean ± SEM. ^※※P < 0.01 vs. group N; ^#P < 0.05 vs. group H + AAV-Control. N normoxia for 4 weeks, H4 hypoxia for 4 weeks, H + AAV-Control transfection of AAV-Control vectors maintained at hypoxia for 4 weeks, H + AAV-CTRP9 transfection of AAV-CTRP9 vectors maintained at hypoxia for 4 weeks.