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. 2021 May 31;11:11343. doi: 10.1038/s41598-021-90596-7

Table 2.

Comparison of haplotypes, genotypes, linkage groups, and the number of activating KIRs between endemic Burkitt lymphoma patients and healthy controls.

Gene eBL n = 104 HC n = 104 OR (95% CI) p-value
N (%) N (%)
Haplotype
A 118 (56.7) 126 (60.6) 0.853 (0.487–1.487) 0.673
B 90 (43.3) 82 (39.4)
KIR haplogroup and genotype frequencies
AA 29 (27.9) 36 (34.6) 0.730 (0.400–1.308) 0.370
Bx 75 (72.1) 68 (65.4)
AB 60 (57.7) 54 (51.9) 1.263 (0.733–2.190) 0.486
BB 15 (14.4) 14 (13.5) 1.083 (0.511–2.297) 1.000
KIR Bx subgroup (Linkage group) frequencies
C4T4 4 (3.8) 0 NA NA
CxT4 10 (9.6) 7 (6.7) 1.474 (0.554–3.818) 0.614
C4Tx 14 (13.5) 17 (16.3) 0.796 (0.386–1.716) 0.698
CxTx 76 (73.1) 80 (76.9) 0.814 (0.431–1.510) 0.631
C4 gene-cluster 18 (17.3) 17 (16.3) 1.071 (0.533–2.178) 1.000
T4 gene-cluster 14 (13.5) 7 (6.7) 2.156 (0.807–5.495) 0.166
Number of activating KIRs
≥ 4 25 (24.0) 9 (8.7) 3.340 (1.530–7.825) 0.004
< 4 79 (76.0) 95 (91.3)

p ≤ 0.05 are considered statistically significant; based on the two-tailed Fisher's exact test. The haplotype A and B were obtained as follows; haplotype A = 2NAA + NAB/2n and haplotype B = 2NBB + NAB/2n. The NAA, NAB, and NBB are the numbers of AA, AB, and BB genotypes, n = total number of individuals37, 43.