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. 2021 Jan 20;44(6):921–930. doi: 10.1007/s00270-020-02751-8

Table 3.

Uni- and multivariate analysis of risk factors for appearance or development of PVST after PSE in the 67 patients

Variables No. of patients No. without appearance or growth of PVST after PSE No. with appearance or growth of PVST after PSE Univariate analysis Multivariate analysis
p value OR (95% CI) p value OR (95% CI)
Age 0.3056
Sex 1.0000 0.9 (0.237–3.579)
 Male 46 40 6
 Female 21 18 3
Platelet count 0.0507 0.2663 0.752 (0.455–1.243)
Prothrombin time 0.5911
Total bilirubin 0.5518
Albumin 0.4868
ALT 0.4203
Hepatic encephalopathy 1.0000 0.512 (0.026–10.041)
 Presence 5 5 0
 Absence 62 53 9
ALBI score 0.5161 0.0725 47.820 (0.702–3235.2)
Hepatocellular carcinoma 0.4465 2.292 (0.158–16.82)
 Presence 4 3 1
 Absence 63 55 8
Occlusion or thrombus of the portal venous system before PSE 0.0601 4.444 (1.15–17.22) 0.0539 10.665 (0.962–118.29)
 Presence 24 18 6
 Absence 43 40 3
Pre-PSE maximum diameter of the splenic vein 0.0076 0.0409 1.535 (1.018–2.315)
Pre-PSE total splenic volume 0.1528
Infarcted splenic volume 0.0216 0.0886 0.995 (0.989–1.001)
Infarcted splenic percentage 0.0171 0.0230 1.136 (1.018–1.267)
Steroid used for pain management 0.4661 2.3 (0.487–11.6)
 Used 25 23 2
 Not used 42 35 7
Heparin used after PSE 0.1249 0.275 (0.057–1.208) 0.1336 10.746 (0.483–239.20)
 Used 10 7 3
 Not used 57 51 6

PVST portal venous system thrombosis; PSE partial splenic artery embolization; ALT alanine aminotransferase; ALBI albumin-bilirubin;