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. 2021 Jun 2;11:11659. doi: 10.1038/s41598-021-91293-1

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Salinity induced onset of endoreduplication in Arabidopsis thaliana is mediated by SOG1. Increasing salinity induces replication stress via oxidative modifications of bases, resulting into accumulations of DSBs. ATR-SOG1 and ATM-SOG1 pathways are activated following replication stress and accumulation of DSBs and regulate the expression of various regulators of cell cycle progression and endoreduplication including, CDKB1;1, CDKB2;1, CYCB1;1, CCS52A, WEE1 and E2Fa, resulting in entry into the endocycle. The figure has been created with BioRender.com.