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. 2021 Jun 4;10:35. doi: 10.1186/s40164-021-00227-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Bioinformatics analysis indicated that YTHDF1 was high-expressed in HCC and was correlated with poor survival. A GEPIA and B UALCAN two databases were used to analyze the mRNA level of YTHDF1 in HCC samples and normal liver samples. C LinkedOmics and D UALCAN two databases were used to analyze the mRNA level of YTHDF1 in different stages of HCC. E The mRNA level of YTHDF1 in different grades of HCC was analyzed in UALCAN database. F LinkedOmics, G GEPIA, and H Kaplan–Meier Plotter databases were used for survival analysis to evaluate the influence of YTHDF1 on the overall survival of HCC patients. *P < 0.05, **P < 0.01, ****P < 0.0001