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. 2021 Jun 4;20:100103. doi: 10.1016/j.mcpro.2021.100103

Table 2.

Selected host proteins in infection

Primary process Gene/complex/family Protein name PPI? Abundance? Phosphorylation? Act.? Function in infection (known and/or hypothesized) Cell location Selected SARS-CoV-2 proteomics references
Host cell entry ACE2 Angiotensin-converting enzyme 2 S +/− Virus receptor PM (41, 46, 47, 48, 49, 57, 61, 74, 86)
TMPRSS2 Transmembrane protease serine 2 S Cleaves S (“priming”), especially at S2' site PM (41, 48, 49, 61)
CTSB Cathepsin B S? Cleaves S, alternative to TMPRSS2 EN (41, 46, 78, 112)
CTSL Cathepsin L S Cleaves S, alternative to TMPRSS2 EN (41, 46, 49, 61, 112)
Endosomal release PIKFYVE 1-phosphatidylinositol 3-phosphate 5-kinase Endosome maturation, with TPC2 EN (101)
Protein expression NUP98 Nuclear pore complex protein Nup98 Orf6 + Prevent host nuclear mRNA export NM (56, 101)
LARP1 La-related protein 1 N - Prioritize virus protein expression CP, NU (56, 101, 112)
UPF1 Regulator of nonsense transcripts 1 N Binding by N represses NMD? CP, NU (56, 112)
EIF4H Eukaryotic translation initiation factor 4H Nsp9 Cap-dependent mRNA translation PN (56)
EIF4E2 Eukaryotic translation initiation factor 4E type 2 Nsp2 Represses cap-dependent translation CP (56, 112)
Sec61 complex SEC61 channel-forming translocon complex Nsp8 Protein entry into endoplasmic reticulum CP, ERM (56, 57)
BRD4 Bromodomain-containing protein 4 E + Interference with antiviral response? NU (48, 56)
Protein processing FURIN Furin S Cleaves S (“priming”), especially at S1/S2 site Golgi
Protein degradation CUL2 Cullin 2 Orf10 + Increase degradation of restriction factors? CP, NU (56, 59)
Cell signaling CDK Cyclin-dependent kinase Cell cycle arrest, S/G2 NU, MT, CP (101)
MAPK Mitogen-activated protein kinase + + Viral replication+, stress response NU, CP, MT (59, 101, 102, 122)
AKT RAC-alpha serine/threonine-protein kinase + + −/+ Viral replication+, cell proliferation & apoptosis regulation NU, CP (PM) (59, 101)
Cell structure PHB complex Prohibitin complex nsp2 Signaling interference, mitochondrial antiviral signaling, apoptosis− MT, NU, CP, PM (56, 112, 129)
CK2 complex Casein kinase II N + Cytoskeleton changes, filopodia+ CP, NU (56, 101, 102)
Stress, immunity HSPA5 Endoplasmic reticulum chaperone BiP Unfolded protein response, virus receptor? CP, PM (61)
NKRF NF-kappaB–repressing factor (nsp10) IL-8 induction NO, NU, CP (78)
CFB Complement factor B −/+ + Alternative complement pathway factor Secreted (120, 121, 122)
CFD Complement factor D Activates complement-dependent killing Secreted (120, 123)
CFI Complement factor I + + Prevented from modulating complement Secreted (120, 123)
CFH Complement factor H +/− + Prevented from modulating complement Secreted (120, 122, 123)

Abbreviations: Cell location—CP, cytoplasm; EN, endosome; ER, endoplasmic reticulum; ERM, ER membrane; LY, lysosome; MT, mitochondria; NM, nuclear membrane; NO, nucleolus; NU, nucleus; PM, plasma membrane; PN, perinuclear; other—HS, heparan sulfate; MAVS, mitochondrial antiviral signaling; NMD, nonsense mediated decay.

See supplemental Table S1 for more information on these proteins, complexes, and families. NKFR = nsp10 PPI may not be direct.