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. 2021 Jun 7;11:344. doi: 10.1038/s41398-021-01362-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Cer ceramides, SM sphingomyelins, ChE cholesteryl esters, DG diglycerides, TG triglycerides, PC phosphatidylcholines, LPC lyso-phosphatidylcholines, PE phosphatidylethanolamines, PI phosphatidylinositols. Dots in the plot represent the residuals of group lipids in each participant. Residuals were obtained after regression out age, sex, BMI, diabetes status, hypertension status, medication status for hypertension, and hyperlipidemia, APOE e4 carrier status, education, and current smoking status. T-tests were used to compare the lipid differences between AD and controls. ns: no significance, *p < 0.05.