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. 2021 Jun 6;27:57. doi: 10.1186/s10020-021-00325-z

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig.5 — The effect of HMGB1 on BMDM from WT and CD36^−/− mice. BMDM were purified and treated with HMGB1 for various duration. The expression of p-Erk, total Erk, p-AKT and total AKT was examined by Western blot. The culture supernant IL-1β and IL-6 levels were measured from BMDM treated with HMGB1 for 48 h. *P < 0.05, **P < 0.01. N = 4 per group