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. 2021 May 25;17(5):e1008985. doi: 10.1371/journal.pcbi.1008985

Fig 5. DPX task performance of neurotypical and divergent agents.

Fig 5

A. Raster plots of a neurotypical agent’s pyramidal cell firing, which successfully maintained representations of the ‘B’ cue and ‘X’ probe (Perception: NMDAg,pyr = 0.37 μS, NMDAg,int = 0.30 μS; Memory: NMDAg,pyr = 0.37 μS, NMDAg,int = 0.35 μS). B. Raster plots of a trial in which the activity bump representation in the memory network collapsed during the ISI (-8%—Perception: NMDAg,pyr = 0.3404 μS, NMDAg,int = 0.30 μS; Memory: NMDAg,pyr = 0.3404 μS, NMDAg,int = 0.35 μS). C. Raster plot of a trial in which the activity bump representation in the perception network fails to track current stimuli (+8%—Perception: NMDAg,pyr = 0.37 μS, NMDAg,int = 0.324 μS; Memory: NMDAg,pyr = 0.37 μS, NMDAg,int = 0.378 μS). D. How an exogenous NMDA antagonist, like ketamine, may affect the rate of DPX task errors. E. Increasing error rates of the different trial types as the pyramidal NMDA g was systematically changed. F. Increasing error rates of the different trial types as the interneuron NMDA g was systematically altered.