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. 2021 May 29;24(2):542. doi: 10.3892/mmr.2021.12181

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Copyright: © Chang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Effect of JNK and p38 inhibitors on the viability of HCT116 and SW620 cells. The two cell types were pretreated with JNK inhibitor (SP600125) and p38 inhibitor (SB203580) for 2 h, and then treated with 250 µg/ml RPMTG for 24 h. (A) MTT assay revealed the effects of the JNK inhibitor (SP600125) and p38 inhibitor (SB203580) on the viability of HCT116 cells. (B) MTT assay revealed the effects of the JNK inhibitor (SP600125) and p38 inhibitor (SB203580) on the viability of SW620 cells. Data are expressed as the mean ± SD of three independent experiments. **P<0.01 and ***P<0.001 vs. control group; ^#P<0.05, ^##P<0.001 vs. RPMTG group. RPMTG, total saponins from Rhizoma Panacis Majoris; JNK, c-Jun N-terminal kinase.