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. 2021 Apr 24;187(2):363–374. doi: 10.1007/s10549-021-06229-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Apoptotic cell death caused by fluvastatin is mediated via FAS-FASL pathway. a Western blotting images showing an upregulation in cleaved PARP by fluvastatin treatment to MCF10.AT1 and MCF10.NeoT cells that were completely rescued by GGPP and MVA, but not squalene. b Bar diagram showing an increase in the FASL transcript levels that are normalized to ribosomal protein L19 (RPL19) in the fluvastatin-treated group (10 mg/kg/day) relative to vehicle control group after 16 weeks of treatment. Values represent mean ± SEM, p = 0.06