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. 2021 Jun 10;11:12287. doi: 10.1038/s41598-021-91292-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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High levels of HGK predict a poor prognosis in PCa patients. (A) Kaplan–Meier curves showing the difference in BCR-free survival between patients with high and low expression levels of MAP4K4. (B) Kaplan–Meier curves showing the difference in BCR-free survival between hormonal-therapy treated patients with high and low expression levels of MAP4K. (C) MAP4K4 expression in hormonal-therapy resistant and sensitive PCa patients (TCGA, cutoff for resistance: relapse before 40 months, n = 32). (D) Schematic summarizing of the regulation of Focal Adhesion (FA) turnover and its impact on cellular motility by HGK.