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. 2021 May 28;22(2):811. doi: 10.3892/etm.2021.10243

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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NRG pretreatment alleviates I/R-accelerated inflammatory reactions, oxidative stress and infarct size. The expression levels of (A) TNF-α, (B) IL-1β and (C) IL-6 (n=5); (D) The level of MDA (n=5). (E) The level of SOD (n=5). (F) Representative images of 2,3,5-triphenyltetrazolium chloride stain. (G) Quantitative analysis of infarct size (n=3). NRG, naringin; I/R, ischemia reperfusion; TNF-α, tumor necrosis factor-α; IL, interleukin; MDA, malondialdehyde; SOD, superoxide dismutase; NRG100, 100 mg/kg naringin; LY, PI3K/Akt inhibitor LY294002. ^aP<0.05 vs. sham group; ^bP<0.05 vs. I/R group; ^cP<0.05 vs. NRG100 + I/R group.