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. 2021 May 28;22(2):811. doi: 10.3892/etm.2021.10243

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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NRG pretreatment represses myocardial autophagy through PI3K/Akt signaling cascades. (A) Representative images of western blotting. Quantitative analysis of the (B) Bax, and (C) Bcl levels and the (D) cleaved caspase: caspase 3 and (E) p-Akt: Akt ratio and the (F) beclin-1 and (G) LC3BII: LC3BI ratio (n=3). NRG, naringin; I/R, ischemia reperfusion; LY, PI3K/Akt inhibitor LY294002; NRG100, 100 mg/kg naringin; p-Akt, phosphorylated Akt; LC3B, microtubule-associated protein 1B-light chain 3. ^aP<0.05 vs. sham group; ^bP<0.05 vs. I/R group; ^cP<0.05 vs. NRG100 + I/R group.