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. 2021 May 28;14:639145. doi: 10.3389/fnmol.2021.639145

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Copyright © 2021 Kuo, Wang, Wu, Chen and Tseng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Valproic acid suppresses IL-6 production in the peri-infarct cortex at 24 h after dMCAo surgery. (A,B) IL-1β concentrations in the peri-infarct cortex (A) and ipsilateral hippocampus (B) were measured by ELISA. (C,D) TNF-α concentrations in the peri-infarct cortex (C) and ipsilateral hippocampus (D) were measured by ELISA. (E,F) IL-6 concentrations in the peri-infarct cortex (E) and ipsilateral hippocampus (F) were measured by ELISA. (G,H) IL-10 concentrations in the peri-infarct cortex (G) and ipsilateral hippocampus (H) were measured by ELISA. ***p < 0.001 indicates comparison with the dMCAo + vehicle group with two-way ANOVA, Bonferroni’s post hoc test. The data represent mean ± SEM.