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. 2021 Jun 13;22:180. doi: 10.1186/s13059-021-02402-2

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — m⁶A of processed pseudogenes disrupt their crosstalk with their cognate protein-coding genes. a, b Scatter plots showing the correlation of the expressions of two representative processed pseudogenes DSTNP2 (a) and NAP1L4P2 (b) with their cognate protein-coding genes among 462 B-lymphoblastoid cell lines. P values of Pearson correlations are indicated at the top left corners. c, d Plot of cumulative fraction of correlation coefficients of gene expressions for processed (c) and unprocessed (d) pseudogenes with their cognate protein-coding genes among 462 B-lymphoblastoid cell lines. Pseudogenes with high, medium, and low m⁶A levels are shown separately. Two-tailed Wilcoxon P values are indicated at the bottom right corners