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. 2021 Jun 14;11:12456. doi: 10.1038/s41598-021-89531-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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(A) IPF-associated disease network showing regulation of known pathophysiological players deregulated in the bulk RNA-Seq (GSE92592) of IPF (n = 20) vs control (n = 19). Nodes represent an interaction partner, edges represent interactions; color scaling shows the deregulation in IPF (red: upregulation, blue: downregulation). (B) Extract of annotated RBPs (GO:0003729 = mRNA binding) gene expression in the two independent human IPF datasets (GSE92592, GSE53845). (C) QKI-interactome (45 nodes, 87 edges) illustrating interaction partners around QKI and its regulation in a high-throughput RNA-Seq data of human IPF. Nodes represent an interaction partner, edges represent interactions; color scaling shows the deregulation in IPF (red: upregulation, blue: downregulation).