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. 2021 Jun 3;12:690535. doi: 10.3389/fphar.2021.690535

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Copyright © 2021 Wang, Zhao, Zhao, Yin, Huang, Cheng, Wang, Liu, Pan, Sun, Wang and Zhu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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FGF1^ΔHBS inhibited ADR-induced renal remodeling and dysfunction. (A) Urine albumin-to-creatinine ratio (UACR). (B) Blood urea nitrogen (BUN) levels. (C) Representative images of hematoxylin and eosin (H&E) staining, Sirius red staining, Masson’s trichrome staining, and transmission electron microscopy (TEM) images of renal tissues. (D) Quantification of mesangial expansion. (E) Quantification of the fibrotic area in Sirius red staining. (F) Quantification of the fibrotic area in Masson staining. (G) Hydroxyproline content in renal tissues. (H) Quantification of podocyte foot process effacement. Data are presented as the mean ± SEM (n = 6); *p < 0.05, **p < 0.01, ***p < 0.001.