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The expression of MST4, p-AKT, Snail1, E-cadherin and Ki67 in human clinical HCC tissue specimens. (A-B) Representative images (A) of immunohistochemical staining for MST4, p-AKT, Snail1, E-cadherin and Ki67 in HCC tissues, and statistical analysis across 325 HCC specimens (B). (C) A proposed model of MST4 regulating the PI3K/AKT/Snail1 signaling pathway to modulate EMT, invasion and metastasis in HCC cells.
