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. Author manuscript; available in PMC: 2021 Jun 21.
Published in final edited form as: Cell Physiol Biochem. 2017 Feb 3;41(2):555–568. doi: 10.1159/000457877

Fig. 5.

Fig. 5.

CD38 gene deficiency or chloroquine (chlo) treatment provokes dedifferentiation phenotype switching and proliferation in coronary arteries of mice. (A) Representative western blot gel documents, (B and C) summarized densitometric data and (D) representative confocal microscopic fluorescent images showing relative abundance of vimentin to calponin or α-SMA in coronary arteries of CD38+/+ or CD38−/− mice under ctrl or chlo treatment. (E) Representative image of hematoxylin-eosin staining of paraffin sections of heart indicating the wall thickness of coronary arteries in CD38+/+ and CD38/− mice fed normal diet (ND) or a high-fat Western diet (WD) for 10 weeks or upon chlo treatment. n=5, *P<0.05 vs. CD38+/+ ctrl.