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. 2021 Jun 21;4:765. doi: 10.1038/s42003-021-02283-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a GCPIF4 in the WT background (purple columns) stimulates hypocotyl elongation of independent GCPIF4/WT lines relative to the WT. b GCPIF4 in the pif4-mutant background (orange columns) stimulates hypocotyl elongation of independent GCPIF4/pif4 lines. c Hypocotyl lengths of independent lines expressing HY5 in the guard cells (GCHY5/hy5) or globally under the 35S promoter (35SHY5/hy5) in the hy5-mutant background (light-blue columns). Data points for (a–c) are means ± SE (n = 20–25 for a; n = 15–25 for b; n = 13–25 for c). a, b Asterisks indicate significant differences relative to the WT (Dunnett’s test, P < 0.05). c Different letters indicate a significant differences (Tukey’s HSD test, P < 0.05). d The illustration indicates the gene expressed in guard cells (GCHY5 or GCPIF4) and the genomic background (hy5, WT/pif4). The blue and purple arrows indicate the hypocotyl-growth response stimulated by GCHY5 and GCPIF4, respectively.