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. 2021 Jun 25;9:30. doi: 10.1186/s40635-021-00393-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Renal endothelial structure. Electron microscopy images of kidney tissue of rats undergoing cardiopulmonary bypass (a, b, CPB, n = 3, with different magnification) and CPB with aprotinin treatment (c, d, CPB + AP, n = 4, with different magnification) and quantification of endothelial vacuolization (white triangle, e), membrane blebbling (black star, f), renal endothelial gap formation (black arrow, G) and endothelial cell detachment from basement membrane (white arrow, h). Data are presented as mean ± standard deviation, ***p < 0.0005, ****p < 0.0001 CPB vs. CPB + AP