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. 2021 Jun 18;13(6):2085. doi: 10.3390/nu13062085

Figure 2.

Figure 2

Maternal EtOH exposure: effects on their offspring related to reproductive parameters, selenium homeostasis, hepatic oxidation and metabolic changes. All the results are extracted from a chronic EtOH exposure model applied to mothers during preconception, gestation and lactation periods carried out by this Research Group, except the data included in the metabolic changes section: Fetal Programming Effects. FFI: fertility female index (number of pregnancies/number of mating × 100), GI: gestation index (number of successful births/number of pregnancy rats × 100), LSI: lactation survival index ((number of total offspring—number of died offspring/number of total offspring) × 100), IUGR: intrauterine growth retardation, LP: lactating pups, GSH: glutathione, GPx: glutathione peroxidase, CAT: catalase, GR: glutathione reductase, SelP: selenoprotein P, Chol: cholesterol, TG: triglycerides, OS: oxidative stress, GFR: glomerular filtration rate, IR: insulin resistance, NALFD: non-alcoholic fatty liver disease, HPA: hypothalamic–pituitary–adrenal axis, IGF-1: insulin-like growth factor, RAS: renin-angiotensin system.