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. 2021 Jun 26;40:216. doi: 10.1186/s13046-021-02018-1

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Correlated expression of DLEU2 and its downstream signaling molecules in EC. a Genomic profiling of DLEU2 and its downstream signaling molecules in human TCGA EC tissues determined by cBioPortal analysis. b qRT-PCR analysis of the indicated genes and miRNAs in EC and adjacent normal tissues. c Correlation of DLEU2 levels with the expression of its downstream signaling molecules in EC tissues as assessed using qRT-PCR analysis. d A scheme depicting how DLEU2 induces EMT and sustains glycolysis in EC via activating the HK2 signaling by competitively binding with miR-455 and by modulating the EZH2/miR-181a pathway. *P < 0.05