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. 2021 Jun 26;21:610. doi: 10.1186/s12879-021-06226-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Longitudinal analysis of HBV DNA VL in chronic HBV patients with TDF treatment vs. without treatment. (A) Distribution of HBV DNA VL at each time point; (B) Stratification of HBV DNA VL at each time point; (C) Kaplan-Meier analysis to estimate the probability of patients with HBV DNA VL > 20 IU/ml in each group over time. In Kaplan-Meier analysis, real dates were used for calculating the time to HBV DNA VL suppressed < 20 IU/ml, rather than the imputed time points and censored subjects were marked on the curve by dots. * p-value < 0.05, ** p-value < 0.01, *** p-value < 0.001, **** p-value < 0.0001. HBV, hepatitis B virus; TDF, Tenofovir disoproxil fumarate; VL, viral load