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. 2021 May 8;147(8):2271–2280. doi: 10.1007/s00432-021-03656-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Integrated analysis of PD-L1 expression and molecular genetic hallmarks of glioma. Analysis of PD-L1 expression and IDH status showed higher expression of PD-L1 in IDH wild type compared to IDH R132H mutated glioma (a). In case of TERT promoter mutation there was higher PD-L1 expression in TERT C228T and C250T mutated glioma (b). Analysis of LOH 1p/19q showed higher expression of PD-L1 in gliomas without LOH 1p/19q (d). Analysis of MGMT promoter methylation showed higher expression of PD-L1 in methylated glioma (c). Indicated are mean and SEM. m methylated, u unmethylated; n.s. not significant (p > 0.05), *p < 0.05