Table 3.
Association between urgency allocation, diagnosed ACS and other LTEs
| Women | ACS n=85 (8.6%) |
No ACS n=908 (91.4%) |
P value 1* | P value 2† |
| U1 | 62 (72.9) | 371 (40.9) | <0.001 | <0.001 |
| U2 | 12 (14.1) | 231 (25.4) | ||
| U3–U5 | 11 (13.0) | 306 (33.6) | ||
| Men |
ACS n=120 (15.0%) |
No ACS n=682 (85.0%) |
||
| U1 | 84 (70.0) | 290 (42.5) | <0.001 | <0.001 |
| U2 | 19 (15.8) | 142 (20.8) | ||
| U3–U5 | 17 (14.2) | 250 (36.7) | ||
| Women |
ACS or LTE n=107 (10.8%) |
No ACS or LTE n=886 (89.2%) |
||
| U1 | 71 (66.4) | 362 (40.9) | <0.001 | <0.001 |
| U2 | 21 (19.6) | 222 (25.1) | ||
| U3–U5 | 15 (14.0) | 302 (34.0) | ||
| Men |
ACS or LTE n=143 (17.8%) |
No ACS or LTE n=659 (82.2%) |
||
| U1 | 96 (67.1) | 278 (42.2) | <0.001 | <0.001 |
| U2 | 24 (16.8) | 137 (20.8) | ||
| U3–U5 | 23 (16.1) | 244 (37.0) |
*P value 1: U1 vs U2, U3, U4 and U5.
†P value 2: U1, U2 vs U3, U4, U5.
‡LTEs consist of ACS, pulmonary embolism, thoracic aortic dissection, acute heart failure, stroke, abdominal aortic aneurysm, severe COPD exacerbation, diabetic ketoacidosis, coronary spasm probably caused by hypokalaemia, epileptic insult, bleeding from oesophageal varices, ovarian torsion and ventricular fibrillation.
ACS, acute coronary syndrome; LTE, life-threatening event.