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. 2021 Jun 28;12(7):656. doi: 10.1038/s41419-021-03938-8

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — A The CCK-8 assays, and colony formation assays (B), EdU assays (C), and transwell assay (D) were used to evaluate the cell growth or invasiveness after transfection with sh-NC, sh-circ-0005105, miR-20a-3p inhibitor, pcDNA3.1-COL11A1, or co-transfected with sh-circ-0005105 and miR-20a-3p inhibitor or pcDNA3.1-COL11A1 in pancreatic cancer cells. A mechanism diagram depicting that the circ-0005105/ miR-20a-3p/COL11A1 axis affects the progression of pancreatic cancer through activating EMT. The results are presented as the mean ± SD for each group. *P < 0.05, **P < 0.01, ***P < 0.001.