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(A) The involvement of vitamin E in the inactivation of 15‐lipoxyganse through the reduction of Fe3+ to Fe2+ leading to ferroptosis prevention. (B) Involvement of oxygen and ferric iron in induction of lipid hydroperoxides leading to ferroptosis. (C) inhibition of ferroptosis using vitamin E leading to elimination of injuries to the heart, kidney, nervous system, liver, gastrointestinal, and lung
