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. 2017 Sep 9;24(1):1273–1283. doi: 10.1080/10717544.2017.1370620

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© 2017 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — (A) The SD-OCT examination suggested the retinal architecture of MNU group was significantly devastated by MNU administration. The retinal thickness of MNU group was significantly smaller than the normal controls. The retinal thickness of the EPO treated group was smaller than the normal controls. However, the retinal thickness of the EPO treated group was larger than that in the MNU group. The retinal thickness of the normal + EPO group was not significantly different from the normal controls. (B) The ONL of the MNU group disappeared after MNU administration. Conversely, a large proportion of ONL was retained in the retinas of EPO treated group. The mean ONL thickness of the EPO treated group was significantly smaller than the normal controls. However, the mean ONL thickness of the EPO treated group was significantly larger than the MNU group. The mean ONL thickness of the Normal + EPO group was not significantly different from the normal controls (#p < .01, for differences compared between animal groups; all the values were presented as mean ± SD; ONL: outer nuclear layer; OPL: outer plexiform layer; INL: inner nuclear layer; IPL: inner plexiform layer; GCL: ganglion cell layer).