Table 1.

Possible self-antigens homologous to B cell epitopes of SARS-CoV-2 (Minota et al., 1988; Mantej et al., 2019; Margutti et al., 2008; Labrador-Horrillo et al., 2014; Shi et al., 2015; Bartos et al., 2007; Oh et al., 2013; Fialová et al., 2013; Takahashi et al., 2010; Kurasawa et al., 2010; Oliveira et al., 2013; Nauert et al., 1997; Sasaki et al., 2001; Saxena et al., 1994; Mimouni et al., 2004; Foedinger et al., 1995; Hedstrand et al., 2001; Tsouris et al., 2020; Yamamoto et al., 2001; Sköldberg et al., 2002; Stojanov et al., 1996; Mygland et al., 1992; Rogers et al., 1994; Ooka et al., 2003; Landegren et al., 2015; Hagberg et al., 2015; Tsuruha et al., 2001; Maciejewska-Rodrigues et al., 2010; Shimizu et al., 2019; Lai et al., 2010; Mei et al., 2018).

Footnote:

1. Tissue distribution data was obtained from the human protein atlas (Uhlén et al., 2015).

2. # - Conclusive data for expression patterns of CD94 (Valiante et al., 1997) and LGI-1 (Furlan et al., 2006) is unavailable in the human protein atlas and was sourced from the literature.

3. References cited adjacent to the autoimmune conditions are evidence for demonstrated presence of antibodies against the corresponding human protein.