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. 2021 May;9(9):779. doi: 10.21037/atm-20-6910

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Genomic alterations in patients with HPD, ED, FP, PR, and non-HPD/ED/FP/PR. The histology, gender, and smoking characteristics are summarized at the bottom of the panel. The frequencies on the left of the panel include all alterations for each gene (missense, deletion, amplification, indel, nonsense, frameshift, splice site, fusion, large genomic rearrangement). HPD/ED/FP, n=18; PR, n=5; non-HPD/ED/FP/PR, n=13. FP, fast progression; HPD, hyperprogressive disease; ED, early death; PR, partial response.