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. 2021 Jun 18;8:654766. doi: 10.3389/fmolb.2021.654766

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Copyright © 2021 Li, Luo, Huttad, Zhang, Wang, Feng, Ding and Han.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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RIPK4 inhibits the metastasis of HCC by STAT3 signaling pathway. (A,B) Comparison of Huh7 cell migration and invasion ability of each group by transwell experiment. (C,D) Comparison of Hep3B cell migration and invasion ability of each group by transwell experiment. (E,F) Comparison of Huh7 cell ability of each group by Wound healing assay. (G,H) Comparison of Hep3B cell ability of each group using Wound healing assay. (I) Western blotting analysis of the expression of STAT3 and p-STAT3 and MMP9 and MMP2 protein. ß-actin was used as a internal control. (J) Western blotting analysis of the expression of STAT3 and p-STAT3 and MMP9 and MMP2 protein of Hep3B cell. ß-actin was used as a loading control. The data are expressed as mean ± SD. **p < 0.01; ***p < 0.001.