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. Author manuscript; available in PMC: 2022 Jun 30.
Published in final edited form as: ACS Appl Mater Interfaces. 2021 Jun 18;13(25):29936–29948. doi: 10.1021/acsami.1c06478

Figure 7. Cell membrane disruption by Aβ oligomers (left) and its rescue by ultrasmall MoS2 quantum dots (right).

Figure 7.

While Aβ-o perturbed membrane integrity (left), association of ultrasmall MoS2 QDs and Aβ-o depleted the toxic peptide species in the extracellular space from amyloid aggregation and membrane partitioning, and subsequently prevented elevated actin expression in the cytoskeleton. This facile membrane-centric strategy may prove beneficial for future development of multifunctional AD nanomedicines.