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. 2021 Jul 2;19:196. doi: 10.1186/s12951-021-00939-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Target CNV ability of RGD-PEI/SAA and mPEI/SAA in mice. RGD-PEI/SAA and mPEI/SAA were labeled with FITC and injected (20 µM, 2 µL/eye) into vitreous space immediately after laser-induced CNV. a The RPE/choroid/sclera flat mounts were prepared 72 h after laser injury. The fluorescence in the representative photographs indicates the localization of FITC-labeled RGD-PEI/SAA and mPEI/SAA in the CNV lesions. The white bar represents 100 µm. b The quantitative analysis of RPE/choroid/sclera flat mounts CNV lesions (∗p < 0.05, ****p < 0.0001, ns: no significance)