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. 2021 May 23;78(13):5275–5301. doi: 10.1007/s00018-021-03843-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Cellular function of PLS3. The main function of PLS3 is its role in F-actin-binding and -bundling activity and, thus, in F-actin dynamics. Consequently, PLS3 is involved in endocytosis, cell motility, cell adhesion, mechanotransduction, pathogen infection, Ca²⁺ homeostasis, exocytosis, vesicle trafficking, axonal local translation and others. PLS3 protein is depicted as red circle. ER endoplasmic reticulum, ECM extra cellular matrix. The figure was created with BioRender.com