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. Author manuscript; available in PMC: 2022 May 1.
Published in final edited form as: Eat Disord. 2021 Jan 6;29(3):226–244. doi: 10.1080/10640266.2020.1864587

Table 3.

Phenotypic, additive genetic correlations (rg), and non-shared environmental correlations (re) between PCL-C and TFEQ, along with 95% confidence intervals from AE models.

Phenotypic Correlation with PCL-C total score (95% CI) Proportion of Phenotypic Correlation
rg (95% CI) re (95% CI)
Measure Genetic Effects Environmental Effects
TFEQ Total .32 (.25; .40) 51% 49% .34 (.07; .58) .29 (.11; .45)
 Cognitive Restraint .08 (.00; .21) * * * *
 Uncontrolled Eating .31 (.23; .39) 69% 31% .53 (.24; .84) .16 (−.01; .33)
 Emotional Eating .29 (.22; .34) 39% 61% .27 (−.05; .57) .30 (.12; .45)

Note: PCL-C = PTSD Checklist – Civilian Version; TFEQ = Three Factor Eating Questionnaire-Reduced 18; Cognitive Restraint = TFEQ-R18 Cognitive Restraint subscale; Uncontrolled Eating = TFEQ-R18 Uncontrolled Eating subscale; Emotional Eating = TFEQ-R18 Emotional Eating subscale; CI = Confidence Interval; rg = genetic correlation; re = non-shared environmental correlation*

Not computed due to lack of phenotypic association. Significant correlations in bold.