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. 2021 Jul 5;21:352. doi: 10.1186/s12935-021-02069-6

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Knockdown of SRRM2-AS1 suppressed the COAD cell proliferation, migration and invasion. A The SRRM2-AS1 expression profile in primary COAD cell lines (HT-29, DLD-1, SW1116 and RKO) and normal human colon epithelial cell line (FHC). B Transfection efficiency of SRRM2-AS1 siRNA was determined by PCR. C The proliferative ability of HT-29 and SW1116 cells was determined by CCK-8 assay. Transwell assay was performed to analyze cell migration (D) and invasion (E) in transfected HT-29 and SW1116 cells. Data are presented as means ± SD. **p < 0.01, ***p < 0.001, compared with FHC or si-NC