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. 2021 Jul 6;21:195. doi: 10.1186/s12906-021-03369-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — PL inhibits the migration of CRPC cells. A and B, PL decreases cell adhesion to the extracellular matrix in PC3 and DU145 cells. C and E, PL inhibits PC3 and DU145 cell migration in scratch-wound healing assays. The different time periods of PC3 and DU145 were adopted in scratch wound assay due to the difference migration speed between PC3 and DU145. Scale bar, 100 μm. D and F, Quantification of (C) and (E). G and H, PL inhibits PC3 and DU145 migration in transwell assays. Values represent the mean ± standard deviation (SD) of at least three independent experiments. Statistical significance was calculated using unpaired Student’s two-tailed t tests for A, B, G, H, and two-way analysis of variance (ANOVA) for D and F with the p-values (*p < 0.05, **p < 0.01, ***p < 0.001)