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. 2021 Jul 9;9:84. doi: 10.1186/s40337-021-00420-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — A Left: Brain regions where baseline CBF significantly predicts change in BMI between baseline and post-treatment (1-month post-randomisation). Right: Scatter plot, with trend-line added, illustrating the association between baseline CBF and change in BMI, while controlling for AN type and chronicity i.e., lower baseline right insula CBF is associated with increases in BMI. B Left: Brain regions where baseline CBF significantly predicts change in DASS-21 anxiety between baseline and post-treatment (1-month post-randomisation). Right: Scatter plot, with trend-line added, illustrating the association between baseline CBF and change in anxiety, while controlling for AN type and chronicity i.e., lower baseline left amygdala CBF is associated with a reduction in anxiety. Brain images shown at an uncorrected threshold of p < 0.005, slice numbers are in MNI space