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. 2021 Jun 24;12:669162. doi: 10.3389/fimmu.2021.669162

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Copyright © 2021 Saisorn, Saithong, Phuengmaung, Udompornpitak, Bhunyakarnjanarat, Visitchanakun, Chareonsappakit, Pisitkun, Chiewchengchol and Leelahavanichkul

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Renal ischemia reperfusion injury (I/R) induced more prominent apoptosis at 24 h post-I/R and immunoglobulin G (IgG) deposition at120 h post-I/R in renal glomeruli of lupus prone mice. Characteristics of renal injury from Fcgr2b-/- or wild-type (Fcgr2b+/+) mice after renal ischemia reperfusion injury (I/R) as evaluated by glomerular apoptosis (activated caspase 3) (A), glomerular IgG deposition (B) (n = 6–7/time-point) with the representative immunofluorescent glomerular pictures (activated caspase 3 and IgG deposition) from mice at 0, 24, and 120 h post-I/R (original magnification 630x) (C) are demonstrated.