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. 2021 Jul 9;12(7):690. doi: 10.1038/s41419-021-03960-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A MTT assay showed that knocking down the expression of PSMC2 in breast cancer cells inhibited cell proliferation. B Flow cytometry suggested that PSMC2 knockdown promoted apoptosis of breast cancer cells. C The detection of cell cycle distribution indicated that PSMC2 knockdown induces cell cycle arrest in G2 phase. D Transwell assay displayed the weakened migration ability of breast cancer cells after silencing PSMC2. E The expression of EMT markers including N-cadherin, Snail and Vimentin was decreased in PSMC2 knockdown cells. Data were shown as mean ± SD (n ≥ 3). * P < 0.05, *** P < 0.001.