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. Author manuscript; available in PMC: 2022 Jun 1.
Published in final edited form as: Arthritis Rheumatol. 2021 May 2;73(6):1062–1072. doi: 10.1002/art.41615

Figure 3.

Figure 3.

JM disease signature: comparison with transcriptomic datasets of skin lesions from adult DM and other inflammatory skin diseases. The 3 gene JM transcriptomic signature identified is the highest in juvenile and adult DM compared to other skin disease lesions (GEO GSE142807, GSE46239, GSE81071, GSE130403, and unpublished microarray datasets, courtesy of Dr. Johann Gudjonsson, Department of Dermatology, University of Michigan)(21, 23, 40). DM lesional samples are represented in red. Vertical dashed lines separate the studied datasets. Each dataset had its control sample set. DLE: discoid lupus erythematosus, SCLE: subacute cutaneous lupus erythematosus, HLP: Hypertrophic lichen planus, LP: lichen planus, GVHD: graft versus host disease. cSLE patients who had two biopsies are colored in blue and yellow. * p-value<0.05; ** p-value<0.01; *** p-value<0.0001.