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. 2021 May 17;105(1):117–124. doi: 10.4269/ajtmh.20-1588

Table 1.

Characteristics of study population (n = 243)

Characteristics n (max, min) %
Age (years)* 49 (10, 87)
Male 157 64.6
Fibrosis stage (n = 66)
 F0 (< 5.1 kPa) 3 4.5
 F1 (≥ 5.1 and < 7.2 kPa) 4 6.1
 F2 (≥ 7.2 and < 9.6 kPa) 19 28.8
 F3 (≥ 9.6 and < 14.1 kPa) 20 30.3
 F4 (≥ 14.1 kPa) 20 30.3
History of HCV treatment (n = 48)
 IFN or PegIFN 44 91.7
 DAA§ 7 14.6
 Both IFN/PegIFN and DAA 3 6.3
HCV genotypes (n = 172)
1 108 62.8
 1a 57 33.1
 1b 40 23.3
 Subtype unknown 11 6.4
2 20 11.6
 2a/c 14 8.1
 Subtype unknown 6 3.5
3 26 15.1
 3a 15 8.7
 3k 11 6.4
4 17 9.9
 4h 6 3.5
 Subtype unknown 11 6.4
6 1 0.6
 6c 1 0.6
 Indeterminate 8 4.7
*

Median (minimum, maximum).

By transient elastography (TE) using FibroScan®, cirrhosis was defined as having a TE value of 14.1 kPa or above (fibrosis stage 4) [31].

With previous HCV treatment.

§

Either daclatasvir/sofosbuvir or sofosbuvir/ledipasvir with or without ribavirin.

Genotypes among 180 patients with quantifiable viral load by Roche HCV.