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. 2021 Jun 28;12:662666. doi: 10.3389/fphys.2021.662666

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Copyright © 2021 Jiang, Zhou, Zhou, Zhang, Hu and Bai.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Bariatric surgery inhibited oxidative stress injury through activating PPARα. The expression of PPARα was detected by immunofluorescence (IF; scale bar, 50 μm) (A) and Western blotting (B). (C) Mitochondrial ROS detected by DHR123 staining (scale bar, 50 μm). (D) Fibrosis of the kidney tissues observed with Masson staining (scale bar, 100 μm). (E) Quantification of the oxidation-related factors (SOD, MDA, and MPO) in rat serum by ELISA. (F) Renal apoptosis detected by TUNEL staining (scale bar, 50 μm). All data represented the mean ± SD. *p < 0.05 [compared with the control (Con) group]; ^#p < 0.05 [compared with the diabetic nephropathy (DN) group]. ROS, reactive oxygen species; SOD, superoxide dismutase; MDA, malondialdehyde; MPO, myeloperoxidase.