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. 2021 Jul 13;11:14385. doi: 10.1038/s41598-021-93755-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Impact of GPR55 deficiency on cardiac macrophage expansion following MI. (a) Representative gating for flow cytometric quantification of F4/80+ macrophages and CD206+ as well as MERTK+ populations in WT and GPR55−/− hearts. (b) Quantification of total macrophage counts and subsets 3 days post-MI in WT and GPR55−/− mice. (c) LV Mertk gene expression in WT and GPR55−/− mice 3 days post-MI relative to no MI WT. Bars indicate mean ± SE (n = 5–10/group) (Student’s t-test).