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. 2021 Jul 13;18:71. doi: 10.1186/s12986-021-00596-7

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Asiatic Acid (AA) ameliorated AngII-induced cardiac hypertrophy and fibrosis in vivo. a Heart weight to tibia length ratio of different groups, n = 9. b Representative M-mode echocardiographic tracings of different groups. c Representative wheat germ agglutinin-stained of the left ventricles to cardiomyocyte size and quantification of the cardiomyocyte size in the indictated groups (n = 9 per group). d Representative Masson-staining of the left ventricles to assess cardiac fibrosis and quantification of the fibrosis area in different groups (n = 9 per group). e ANP (atrial natriuretic peptide) and B-MHC protein levels in the heart detected by Western blotting in different groups (n = 6 per group). f The expression of miRNA-126 and pre-miR-126 levels in the hearts of the different groups (n = 7 per group). Data are presented as the mean ± SD, **P < 0.01