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. 2021 Jul 13;12:394. doi: 10.1186/s13287-021-02458-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — IL-6 which increased FOXS1 expression in glioma cells is mainly secreted by CD90^low gaMSCs. IL-6 secretion in conditioned media of gaMSCs, CD90^lowgaMSCs and CD90^highgaMSCs was measured by ELISA (A, C). The effect of IL-6 on FOXS1 expression in glioma cells could be reversed by IL-6 antibody (B). The FOXS1 expression in glioma cells is higher in conditioned media of CD90^lowgaMSCs than that of CD90^highgaMSCs (D). (n ≥ 3, *P < 0.05, **P < 0.01, ***P < 0.001, ****P < 0.0001)